http://www.thaindian.com/newsportal/health1/container-carrying-swine-flu-virus-explodes-on-swiss-train_100185502.html Train explosion switzerland carrying virus container http://www.washingtonpost.com/wp-dyn/content/article/2005/10/09/AR2005100900932.html Army digs 1918 body spanish flu virus victim threes year ago or so my time frame maybe off but does it really matter for you sceptics http://onlinejournal.com/artman/publish/article_4631.shtml http://www.cnn.com/2009/US/04/22/missing.virus.sample/index.html missing and or stolen flu virus http://uk.reuters.com/article/governmentFilingsNews/idUKN2445216420090424?pageNumber=2&virtualBrandChannel=0 pharamacy company jumping for joy our friends fromwikea and sooo Cytokines (Greek cyto-, cell; and -kinos, movement) are a category of signaling molecules that are used extensively in cellular communication. They are proteins, peptides or glycoproteins. The term cytokine encompasses a large and diverse family of polypeptide regulators that are produced widely throughout the body by cells of diverse embryological origin.[1] Historically, the term "cytokine" has been used to refer to the immunomodulating agents (interleukins, interferons, etc.). Conflicting data exists about what is termed a cytokine and what is termed a hormone. Anatomic and structural distinctions between cytokines and classic hormones are fading as we learn more about each. Classic protein hormones circulate in nanomolar (10^-9) concentrations that usually vary by less than one order of magnitude. In contrast, some cytokines (such as IL-6) circulate in picomolar (10^-12) concentrations that can increase up to 1,000-fold during trauma or infection. The widespread distribution of cellular sources for cytokines may be a feature that differentiates them from hormones. Virtually all nucleated cells, but especially endo/epithelial cells and resident macrophages (many near the interface with the external environment) are potent producers of IL-1, IL-6, and TNF-α. In contrast, classic hormones, such as insulin, are secreted from discrete glands, such as the pancreas (Inflammatory Cytokines in Nonpathological States, Joseph G. Cannon, News Physiol. Sci., Volume 15, December 2000). As of 2008, the current terminology refers to cytokines as immunomodulating agents. However, more research is needed in this area of defining cytokines and hormones. The action of cytokines may be autocrine, paracrine, and endocrine. Cytokines are critical to the development and functioning of both the innate and adaptive immune response, although not limited to just the immune system. They are often secreted by immune cells that have encountered a pathogen, thereby activating and recruiting further immune cells to increase the system's response to the pathogen. Cytokines are also involved in several developmental processes during embryogenesis. Contents [hide] * 1 Effects * 2 Nomenclature * 3 Classification o 3.1 Structural o 3.2 Functional * 4 Cytokine receptors * 5 Cysteine-knot cytokines * 6 References * 7 Further reading * 8 See also * 9 External links [edit] Effects Each cytokine has a matching cell-surface receptor. Subsequent cascades of intracellular signalling then alter cell functions. This may include the upregulation and/or downregulation of several genes and their transcription factors, resulting in the production of other cytokines, an increase in the number of surface receptors for other molecules, or the suppression of their own effect by feedback inhibition. The effect of a particular cytokine on a given cell depends on the cytokine, its extracellular abundance, the presence and abundance of the complementary receptor on the cell surface, and downstream signals activated by receptor binding; these last two factors can vary by cell type. Cytokines are characterized by considerable "redundancy", in that many cytokines appear to share similar functions. Generalization of functions is not possible with cytokines. Nonetheless, their actions may be grouped as: * autocrine, if the cytokine acts on the cell that secretes it. * paracrine, if the target is restricted to the immediate vicinity of a cytokine's secretion. * endocrine, if the cytokine diffuses to distant regions of the body (carried by blood or plasma). It seems to be a paradox that cytokines binding to antibodies have a stronger immune effect than the cytokine alone. This may lead to lower therapeutic doses. Overstimulation of cytokines can trigger a dangerous syndrome known as a cytokine storm; this may have been the cause of severe adverse events during a clinical trial of TGN1412. [edit] Nomenclature Cytokines have been classed as lymphokines, interleukins, and chemokines, based on their presumed function, cell of secretion, or target of action. Because cytokines are characterised by considerable redundancy and pleiotropism, such distinctions, allowing for exceptions, are obsolete. * The term interleukin was initially used by researchers for those cytokines whose presumed targets are principally leukocytes. It is now used largely for designation of newer cytokine molecules discovered every day and bears little relation to their presumed function. The vast majority of these are produced by T-helper cells. * The term chemokine refers to a specific class of cytokines that mediates chemoattraction (chemotaxis) between cells. IL-8 (interleukin-8) is the only chemokine originally named an interleukin. [edit] Classification [edit] Structural Structural homology has been able to partially distinguish between cytokines that do not demonstrate a considerable degree of redundancy so that they can be classified into four types: * The four α-helix bundle family - Member cytokines have three-dimensional structures with four bundles of α-helices. This family in turn is divided into three sub-families: 1. the IL-2 subfamily 2. the interferon (IFN) subfamily 3. the IL-10 subfamily. The first of these three subfamilies is the largest. It contains several non-immunological cytokines including erythropoietin (EPO) and thrombopoietin (THPO). Also, four α-helix bundle cytokines can be grouped into long-chain and short-chain cytokines. * the IL-1 family, which primarily includes IL-1 and IL-18 * the IL-17 family, which has yet to be completely characterized, though member cytokines have a specific effect in promoting proliferation of T-cells that cause cytotoxic effects [edit] Functional A classification that proves more useful in clinical and experimental practice divides immunological cytokines into those that enhance cytokine responses, type 1 ( IFN-γ, TGF-β etc.), and type 2 (IL-4, IL-10, IL-13, etc.), which favor antibody responses. A key focus of interest has been that cytokines in one of these two sub-sets tend to inhibit the effects of those in the other. Dysregulation of this tendency is under intensive study for its possible role in the pathogenesis of autoimmune disorders. [edit] Cytokine receptors Main article: Cytokine receptor In recent years, the cytokine receptors have come to demand the attention of more investigators than cytokines themselves, partly because of their remarkable characteristics, and partly because a deficiency of cytokine receptors has now been directly linked to certain debilitating immunodeficiency states. In this regard, and also because the redundancy and pleiomorphism of cytokines are, in fact, a consequence of their homologous receptors, many authorities think that a classification of cytokine receptors would be more clinically and experimentally useful. A classification of cytokine receptors based on their three-dimensional structure has, therefore, been attempted. Such a classification, though seemingly cumbersome, provides several unique perspectives for attractive pharmacotherapeutic targets. * Immunoglobulin (Ig) superfamily, which are ubiquitously present throughout several cells and tissues of the vertebrate body, and share structural homology with immunoglobulins (antibodies), cell adhesion molecules, and even some cytokines. Examples: IL-1 receptor types. * Haemopoietic Growth Factor (type 1) family, whose members have certain conserved motifs in their extracellular amino-acid domain. The IL-2 receptor belongs to this chain, whose γ-chain (common to several other cytokines) deficiency is directly responsible for the x-linked form of Severe Combined Immunodeficiency (X-SCID). * Interferon (type 2) family, whose members are receptors for IFN β and γ. * Tumor necrosis factors (TNF) (type 3) family, whose members share a cysteine-rich common extracellular binding domain, and includes several other non-cytokine ligands like CD40, CD27 and CD30, besides the ligands on which the family is named (TNF). * Seven transmembrane helix family, the ubiquitous receptor type of the animal kingdom. All G-protein coupled receptors (for hormones and neurotransmitters) belong to this family. Chemokine receptors, two of which act as binding proteins for HIV (CXCR4 and CCR5), also belong to this family. 1918 Spanish Influenza Outbreak: The Enemy Within By Christine M. Kreiser | American History | 3 comments | Print This Post | Email This Post Horse-drawn carts plied the streets with a call to bring out the dead in the city where bodies lay unburied for days. The afflicted died by the thousands, and survivors lived in fear. But this wasn’t medieval Europe being stalked by the Black Death. This was Philadelphia, October 1918, and the city was under siege from a new variant of one of mankind’s oldest specters: influenza. The flu lurking in the midst of this patriotic fervor, however, would prove far more lethal than trench warfare and poison gas. Most alarming was the fact that the disease ravaged previously healthy young adults in their 20s and 30s: the men and women who worked the factories, cleaned the streets, tended the sick — and fought the wars. Many assumed, wrongly, that the flu had originated in Spain, where 8 million fell ill during a wave of relatively mild flu that had swept the globe in the spring of 1918. Because Spain was neutral and its press uncensored during the war, it was one of the few places in Europe where news about the epidemic was being reported. Whatever its origins, the flu was taking a toll on frontline troops. Commander Erich von Ludendorff blamed the disease for the failure of Germany’s major spring offensive.It was a grievous business, he said,having to listen every morning to the chiefs of staff’s recital of the number of influenza cases, and their complaints about the weakness of their troops. Influenza wasn’t Ludendorff’s only obstacle. General JohnBlack Jack Pershing, commander in chief of the American Expeditionary Forces in Europe, pushed relentlessly to build up troop strength. The U.S. Army had fewer than 100,000 soldiers when it entered the war — the general’s plans called for approximately 4 million. The Americans would not simply plug holes in the British and French lines. The AEF would stand alone, and march to victory under the American flag. To do that, Pershing needed more men, more materiel. Always, endlessly, more. Back home, the ramp-up hit a snag. On March 4, 1918, the Army installation at Camp Funston, Kan., reported a single case of flu. Before the end of the month, 1,100 men had been hospitalized, and 20 percent of those men developed pneumonia. Flu spread rapidly among Army camps as troops were rushed through on their way to the front. But the outbreak had subsided by summer, and it looked like the worst was over. It wasn’t. Only a Matter of Hours Camp Devens, 35 miles northwest of Boston, was seriously overcrowded. Built to house 36,000 troops, it contained more than 45,000 in early September 1918. The flu struck there with a suddenness and virulence that had never been seen before.These men start with what appears to be an ordinary attack of LaGrippe or Influenza, and when brought to the Hosp. they very rapidly develop the most vicious type of Pneumonia that has ever been seen, wrote Roy Grist, a doctor at the Camp Devens hospital.Two hours after admission they have the Mahogany spots over the cheek bones, and a few hours later you can begin to see the Cyanosis extending from their ears and spreading all over the face, until it is hard to distinguish the coloured man from the white….It is only a matter of hours then until death comes….We have been averaging about 100 deaths per day….We have lost an outrageous number of Nurses and Drs. Flu victims were wracked by fevers often spiking higher than 104 degrees and body aches so severe that the slightest touch was torture. Cyanosis was perhaps the most terrifying hallmark of the pneumonia that often accompanied this flu. A lack of oxygen in the blood turned one’s skin a bluish-black — leading to speculation that the Black Death had again come calling. Outdoor emergency ward, Brookline, Massacusetts. Library of Congress. Outdoor emergency ward, Brookline, Massacusetts. Library of Congress. While Devens tried unsuccessfully to contain the outbreak, a similar situation was developing at Commonwealth Pier, a naval facility in Boston. Flu was reported there in late August, but the war would not wait. Sailors were shipped out to New Orleans, Puget Sound and the Great Lakes Naval Training Station near Chicago. Josie Mabel Brown was a young Navy nurse living in St. Louis, Mo., when she was called to duty at Great Lakes.There was a man lying on the bed dying and one was lying on the floor, she said of her first visit to a sick ward.Another man was on a stretcher waiting for the fellow on the bed to die….We wrapped him in a winding sheet and left nothing but the big toe on the left foot out with a shipping tag on it to tell the man’s rank, his nearest of kin, and hometown….Our Navy bought the whole city of Chicago out of sheets. There wasn’t a sheet left in Chicago. All a boy got when he died was a winding sheet and a wooden box; we just couldn’t get enough caskets. Three hundred sailors from Boston landed at the Philadelphia Navy Yard on September 7; on the 19th the Philadelphia Inquirer reported that 600 sailors and marines had been hospitalized with the flu. It should have been apparent to city officials that a potential crisis loomed. In Massachusetts the flu had spread rapidly from military encampments to the public at large. Medical practitioners in Philadelphia called for a quarantine, but Wilmer Krusen, director of the city’s Department of Public Health and Charities, declined. There was recent precedent for such action: Quarantines were regularly enacted during a terrifying polio epidemic in 1916. But that was in peacetime. No civilian deaths from flu had been reported locally, and a Liberty Loan parade — perhaps the largest parade Philadelphia had ever seen — was scheduled for the end of the month. A quarantine would only cause panic, and the city would most certainly not meet its quota of war-bond sales. Every American seemingly had a personal stake in winning the war. Even children were eager to do their bit. Anna Milani, who was a child in Philadelphia during the epidemic, remembered the rhyme she and her friends would sing in the street: Tramp, tramp, tramp the boys are marching I spied Kaiser at the door We’ll get a lemon pie And we’ll squash it in his eye And there won’t be any Kaiser anymore The parade stepped off as planned on September 28 with marching bands, military units, women’s auxiliaries and Boy Scout troops. Some 200,000 spectators thronged the two-mile-long parade route in a show of civic pride. Three days later, 635 new civilian cases of flu, and 117 civilian deaths from the disease and its complications, were reported in Philadelphia. Worry is Useless October 1918 was brutal in the City of Brotherly Love. Schools, churches, theaters and saloons were closed. So many Bell Telephone operators were home sick that the company placed notices in city newspapers pleading with the public tocut out every call that is not absolutely necessary that the essential needs of the government, doctors and nurses may be met. Krusen authorized Bell to discontinue service to those making unnecessary calls, and 1,000 customers were eventually cut off. Even if emergency calls did get through, there weren’t enough people to answer them. A quarter of Philadelphia’s doctors and nurses were away serving in the military. Volunteers were called, but many were too sick themselves — or too frightened of contracting the disease — to be of much help. Entire families were stricken, and the prognosis was often grim.My mother called the doctor because the whole family was sick with this flu, said Harriet Hasty Ferrell.And I, being an infant baby, was very sick, to the point that the doctor thought that I would not make it. He told my mother it wasn’t necessary to feed me anymore. Still, there were those who tried to quell panic. An October 6 editorial in the Inquirer advised:Live a clean life. Do not even discuss influenza….Worry is useless. Talk of cheerful things instead of the disease. No amount of happy talk could make the nightmare go away. Between October 12 and October 19, 4,597 Philadelphians died of the flu and related respiratory diseases, and survivors struggled to carry out familiar mourning rituals.We couldn’t go inside the church, one city native remembered.The priest would say Mass on the step, and we would all be congregated outside….They figured maybe outside you wouldn’t catch the germ. Another recalled that her 13-year-old cousin, who was sick with the flu, had to be carried to the cemetery wrapped in a blanket in order to say the traditional Jewish prayers at his mother’s funeral service. Hundreds of unburied corpses posed another serious health risk. Caskets were in such short supply that the J.G. Brill Co., which manufactured trolley cars, donated packing crates to fill the need. The Bureau of Highways used a steam shovel to dig mass graves in a potter’s field. By the end of the month, the Spanish flu had claimed 11,000 victims in Philadelphia and 195,000 nationwide. The tragedy played out with varying degrees of severity across the country. The city of San Francisco, where the flu hit hardest in late October, mandated that gauze masks be worn in public at all times. The mandate was widely followed, though in reality, masks did little to prevent the spread of flu. They were also uncomfortable and inconvenient, and the public would not tolerate them for long. Even officials showed a less than vigilant attitude when the mayor, a city supervisor, a Superior Court judge, a congressman and a rear admiral were photographed at a prizefight sans their protective masks. And there were those who claimed the act was an unconstitutional attack on personal freedom:If the Board of Health can force people to wear masks, said the San Francisco Chronicle,then it can force them to submit to inoculations, or any experiment or indignity. Doctors searched desperately for a cure, or at least a stop-gap measure. But they were on the wrong track. Conventional wisdom held that the flu was caused by bacteria; vaccines to fight bacterial infections, however, had no effect on the disease. (Flu was not identified as a virus until 1933.) The epidemic was a crushing blow to medical science, which had only recently come to be seen as a professional discipline. Government agencies fared no better. Surgeon General Rupert Blue, head of the U.S. Public Health Service, was aware that an outbreak of flu was possible. But in July 1918, he denied a request for $10,000 to be dedicated to pneumonia research, and he made no other preparations. Blue’s first public warning came in mid-September and included such tips as Avoid tight clothes, tight shoes, tight gloves — seek to make nature your ally not your prisoner and Help by choosing and chewing your food well. Congress appropriated $1 million in emergency funding for USPHS; Blue eventually returned $115,000 to the government. Worse still, the government contributed to the national paranoia surrounding all things German. The USPHS officer for northeastern Mississippi planted stories in the local papers that the Hun resorts to unwanted murder of innocent noncombatants….He has [at]tempted to spread sickness and death thru germs, and has done so in authenticated cases. Lieutenant Colonel Philip Doane, head of the Health and Sanitation Section of the Emergency Fleet Corporation, which oversaw U.S. shipyards, theorized that U-boats had delivered German spies to America to turn loose Spanish influenza germs in a theatre or some other place where large numbers of persons are assembled. So persistent was the belief that Germany had somehow launched a biological attack that USPHS laboratories devoted precious time to investigating claims that Bayer aspirin, which was manufactured in the States under a German-held patent, had been laced with deadly flu germs. “Let the curse be called the German plague, declared The New York Times in October.Let every child learn to associate what is accursed with the word German not in the spirit of hate but in the spirit of contempt born of the hateful truth which Germany has proved herself to be. Over There The death toll mounted at home through September and October even as President Woodrow Wilson was faced with General Pershing’s demands for more soldiers. Through the summer, Americans were being sent to Europe at the rate of 250,000 a month. But flu was running rampant on troopships, and those who survived the interminable voyage simply spread the disease to frontline staging areas. Wilson was urged by several advisers not to dispatch additional troops until the epidemic had been contained. The president consulted with his chief of staff General Peyton March, who conceded that conditions on the overseas transports were hardly ideal. He would not, however, concede anything that might stand in the way of winning the war.Every such soldier who has died [on a troopship], said March,just as surely played his part as his comrade who died in France. Wilson relented. The transports continued. Wilson had won a second term in 1916 because he had kept the United States out of the war. Once war was declared in 1917, however, he could not afford to waver in his commitment to seeing the conflict through to Allied victory. To shore up public support, Wilson created the Committee on Public Information a week after declaring war on Germany. (One of its lasting contributions was the Uncle SamI want you recruiting poster.) The CPI’s news division issued thousands of press releases and syndicated features about the war that made their way, often unedited, into newspapers across the country. The CPI also had a pictorial publicity division, an advertising division and a film division. In short, it used every possible media source to influence public opinion. Wilson’s zeal for advancing democratic ideals abroad was secured by his willingness to suppress them at home. Dissent was not tolerated. Under the 1917 Espionage Act, roundly criticized as being unconstitutional, Socialist leaders Eugene Debs and Victor Berger were sentenced to a combined 30 years in prison for their antiwar protests. The act also gave the postmaster general the right to determine what constituted unpatriotic or subversive reading material and ban it from the U.S. mail. The Justice Department authorized the 200,000 members of a volunteer group called the American Protective League to report on suspected spies,slackers who didn’t buy war bonds and anyone who voiced opposition to the government. In this hyper-patriotic atmosphere, fighting the flu came second to winning the war. Public officials, and the public itself, downplayed the seriousness of the silent enemy within and focused on the more tangible enemies of a nation at war. The Germans could be defeated on the battlefield overseas and by surveillance at home. Nothing could stop a disease that immobilized great cities for weeks and carried off hundreds of thousands in the prime of life. And then, it was over. By the end of 1918, deaths from flu and pneumonia nationwide had subsided greatly, and a third wave in the spring of 1919 left far fewer casualties in its wake.In light of our knowledge of influenza and the way it works, explained Dr. Shirley Fannin, an epidemiologist and current director of disease control for Los Angeles County, Calif.,we do understand that it probably ran out of fuel. It ran out of people who were susceptible. Those who survived their exposure to the flu developed immunity to the disease, but not to its lasting consequences. William Maxwell, writer and longtime editor at The New Yorker, was a 10-year-old in Lincoln, Ill., when the flu struck his family, killing his mother.I realized for the first time, and forever, that we were not safe. We were not beyond harm, he remembered eight decades later.From that time on there was a sadness, which had not existed before, a deep down sadness that never quite went away….Terrible things could happen — to anybody. For all the advances in medical science, it is still not clear where the 1918 virus originated, or why it took such a toll on healthy young adults. Flu viruses are extremely adaptable. According to the National Institutes of Health, one new strain of flu appeared in humans between the Hong Kong flu outbreak in 1969 (the last flu pandemic) and 1977. Between 1997 and 2004, five new strains appeared. Modern researchers agree that it is probably impossible to prevent an outbreak of flu, but it is possible to prepare for one — if the public, health officials and government agencies can agree on a plan of action. Today, as in 1918, a global conflict demands an ever-increasing amount of resources. The government has enacted extraordinary measures in the name of national security. And a public health crisis of the magnitude of the 1918 epidemic is almost incomprehensible. After all, it’s only the flu. Hello Jeff - I am making a plea to everyone who reads this, please, please DO NOT TAKE ANY VACCINE THAT IS PURPORTED TO 'PREVENT' THIS FLU. Remember 1976 and the so called Swine Flu outbreak that was purported to be a coming pandemic? It only infected recruits at Ft. Dix. Why? Because I believe that the so called Swine Flu virus infected the recruits due to the vaccines they were given. Whether the government developed the Swine Flu 1976 virus and infected the recruits as a means to test the public to see if people would comply with a call to take vaccination against Swine Flu, or the recruits became infected via contaminated vaccine they were given as part of the recruit regimen, that outbreak was as phony as they come. I was one of the people duped into taking a Swine Flu shot and it made me so sick. I was sick in bed for three months after taking the vaccine. Do not take seasonal flu vaccine if you are told that it could help prevent this brand new Swine Flu variant. It won't do a thing to prevent this flu. What it will do is serve up new genetic material to the Swine Flu virus that I have dubbed Spanish Flu 2, the Sequel. The Spanish Flu variant will use the gene sequences in the vaccine in humans to develop more of the changes that make the virus more readily infect humans. We do not want to give this virus more human genetic material so that it will infect humans more readily person to person. This is what vaccinated individuals do for pandemic strains. There is also a safety issue in any experimental vaccine, much like the one in 1976. Some people even feel that such a vaccine for pandemic strain might require more than one vaccination which could actually be a binary set up. The first shot might just add some genetic code that stays dormant in the body until one gets the second vaccine shot which then serves to only cause infection. It could trigger Guillain-barre syndrome, Typhus or some other condition. An Influenza vaccine does not protect or prevent a person from contracting flu. It is purported to, maybe, prevent some complications of flu and maybe shorten duration. I am not even sure it does that. Personally, I feel the vaccine weakens our immune system and also sickens us due to contaminants in the vaccine. I feel that people can better protect themselves by washing hands often and thoroughly. People should also use protective gloves when out and about during epidemics. Don't be afraid of "looking odd." I would not be ashamed to use a mask and gloves. I see that the Mexicans are using them. A big problem during a pandemic is that these simple supplies will become extremely scarce awfully quickly. Stock up now. Medical supplies. personal hygene supplies and don't forget fido, or any other pet. Once a pandemic hits, it will be too late to stock up. Water, too. We may lose clean water and electric power, so be prepared. So, please, people, DON'T TAKE ANY VACCINES OFFERED. THEY COULD KILL YOU BEFORE ANY VIRUS KILLS YOU. Pat Doyle Patricia A. Doyle DVM, PhD Bus Admin, Tropical Agricultural Economics Univ of West Indies Please visit my "Emerging Diseases" message board at: http://www.emergingdisease.org/phpbb/index.php Also my new website: http://drpdoyle.tripod.com/ Zhan le Devlesa tai sastimasa Go with God and in Good Health Interesting Calif Dept of Health issued a statement that Tamiflu doesnt work for this if you read the above history above ground government may not have known anything about this but you can bet those that were behind the NWO agenda did and they do now http://www.livablefutureblog.com/pdf/Putting_Meat_on_Table_FULL.pdf Read this report, this a government report how they are poisoning our livestock, fruits and vegetables, by inject with bacteria strains antibiotics that we use CDC warned not to do this. We know all about goals 2000 the UN agenda ect to eliminate people how are they going to do this with out going to war world wide they ignored they are also spraying our crops with Animal feces waste says it a cheaper way to go the animal waste run off it allowed to run into the water supply we drink and then they treat this with nitrates ect. For you skeptics out their do you still think the globalist didn't release a low yield bio weapon Do the research of links I provided if you dont think these people are evil why are you in the Militia ? I have done my best to research whats going on if you have problem with it too bad I put out the facts Im not responsible for the conclusions i didn't test this crap I didn't lose 3 vials of Venezuelan Flu I didn't cause a train explosion in Switzerland carrying this virus in a container of dry ice and i'm certainly not responsible for poisoning the world food supply and our own . Okay going on the assumption that this virus is low yield weapon for argument sake let say it is what do we do -Once ill their is no Kwown antibiotic to get rid of it it has to run its course An ounce of prevention is the Key how to prevent keep from spreading further and getting sick yourself. Effective hand washing after all public contact clean all outside door knobs to your home and all surfaces take vitamine C in Large does of about 4000 - 6000 Milligram for children cut it by half Im starting regimen of vitamin C myself tomorrow hand washing should be done with hot soapy water about 2 minutes or as long as it takes to sing happy birthday I am one of the few EMTs that took a NBC warfare response program back when it was offered to me one of the things that occurs with a low yield weapon or any virus as it spread out it begins to break down and dissipates sun lights appears to make it less effective. Im still looking into that though Go to the links I have provided I would have posted everything but it is just too much to post here Opinion our welcome discussion is welcome I wont waste time in silly arguments and please don't flame what you dont understand for those that hide their head in the sand These facts are all not inconclusive as ongoing research is still being done I could use some help in this Implore you at the very minimum read the report on the farm commission its all connected I will be putting out other information as I get it The official story is a man screwed a pig and released the virus I dont believe that story people Mexico do weird stuff but so does europe and the US . This thing spread in a matter of few hours perhaps we need to track the death and illnesses ourselves to get at the truth. Im waiting on a report where one small town has 11 dead in the US